Novel tricyclic pyrazole BRAF inhibitors with imidazole or furan central scaffolds

نویسندگان

  • Dan Niculescu-Duvaz
  • Ion Niculescu-Duvaz
  • Bart M.J.M. Suijkerbuijk
  • Delphine Ménard
  • Alfonso Zambon
  • Arnaud Nourry
  • Lawrence Davies
  • Helen A. Manne
  • Frank Friedlos
  • Lesley Ogilvie
  • Douglas Hedley
  • Andrew K. Takle
  • David M. Wilson
  • Jean-Francois Pons
  • Tom Coulter
  • Ruth Kirk
  • Neus Cantarino
  • Steven Whittaker
  • Richard Marais
  • Caroline J. Springer
چکیده

V-RAF murine sarcoma viral oncogene homolog B1 (BRAF) is a serine/threonine-specific protein kinase that is mutated with high frequency in cutaneous melanoma, and many other cancers. Inhibition of mutant BRAF is an attractive therapeutic approach for the treatment of melanoma. A triarylimidazole BRAF inhibitor bearing a phenylpyrazole group (dimethyl-[2-(4-{5-[4-(1H-pyrazol-3-yl)-phenyl]-4-pyridin-4-yl-1H-imidazol-2-yl}-phenoxy)-ethyl]-amine, 1a) was identified as an active BRAF inhibitor. Based on this starting point, we synthesized a series of analogues leading to the discovery of 6-{2-[4-(4-methyl-piperazin-1-yl)-phenyl]-5-pyridin-4-yl-3H-imidazol-4-yl}-2,4-dihydro-indeno[1,2-c]pyrazole (1j), with nanomolar activity in three assays: inhibition of purified mutant BRAF activity in vitro; inhibition of oncogenic BRAF-driven extracellular regulated kinase (ERK) activation in BRAF mutant melanoma cell lines; and inhibition of proliferation in these cells.

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عنوان ژورنال:

دوره 18  شماره 

صفحات  -

تاریخ انتشار 2010